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Magyar Hypertonia Társaság Gene-Environment Interaction in Health and Disease (ISP 2010) Rendezvény vége: 2010.09.25. Tudományos információ: Pavel Hamet Címe: Canada Munkahely neve: Congress secretariat ISP & SHR 2010 – JPdL Címe: Canada Montréal, Québec 1555, Peel Street, Suite 500 H3A 3L8 Helyszín: A rendezvény Web oldala: http://www.ispmontreal2010.com/ Following the 2006 meeting in Beijing we invite colleagues interested in the pathophysiology of diseases to join us for the 6th International Congress, which will be held in Montréal, Canada from September 22 to 25, 2010, under the banner “Gene-environment interaction in health and disease ”. The Congress program will reflect current trends in the evolution of pathophysiology and biological science in general, while further enlarging the scope of our meeting to cover progress from Genomics to Systems Biology, integrating both our genetic make-up and environmental exposures resulting in shared and distinct pathophysiological pathways. We also plan to present novel therapeutic targets based on this new knowledge, future tools in prevention and optimized therapy based on predictive genomics.
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Magyar Hypertonia Társaság Gene-Environment Interaction in Health and Disease (ISP 2010) Rendezvény vége: 2010.09.25. Tudományos információ: Pavel Hamet Címe: Canada Munkahely neve: Congress secretariat ISP & SHR 2010 – JPdL Címe: Canada Montréal, Québec 1555, Peel Street, Suite 500 H3A 3L8 Helyszín: A rendezvény Web oldala: http://www.ispmontreal2010.com/ Following the 2006 meeting in Beijing we invite colleagues interested in the pathophysiology of diseases to join us for the 6th International Congress, which will be held in Montréal, Canada from September 22 to 25, 2010, under the banner “Gene-environment interaction in health and disease ”. The Congress program will reflect current trends in the evolution of pathophysiology and biological science in general, while further enlarging the scope of our meeting to cover progress from Genomics to Systems Biology, integrating both our genetic make-up and environmental exposures resulting in shared and distinct pathophysiological pathways. We also plan to present novel therapeutic targets based on this new knowledge, future tools in prevention and optimized therapy based on predictive genomics.
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